"Treatment of NSAID toxicosis consists of early decontamination, protection of the GI tract and kidneys, and supportive care. Vomiting should be induced in recent exposures, followed by administration of activated charcoal with a cathartic. Activated charcoal can be repeated in 6-8 hr to prevent NSAID reabsorption from enterohepatic recirculation. Use of H2-receptor antagonists (ranitidine, famotidine, cimetidine) may not prevent GI ulcers but can be useful in treating them. Omeprazole, which is a proton pump inhibitor used for inhibiting gastric acid secretions, can be used instead of an H2-blocker at 0.5-1.0 mg/kg, PO, sid, in dogs. Sucralfate (dog: 0.5-1 g, PO, bid-tid; cat: 0.25-0.5 tablet, PO, bid-tid) reacts with hydrochloric acid in the stomach and forms a paste-like complex that binds to the proteins in ulcers and protects them from further damage. Because sucralfate requires an acidic environment, it should be given ³30 min before administering H2 antagonists. Misoprostol (dog: 1-3 mg/kg, PO, tid) has recently been shown to prevent GI ulceration when used concomitantly with aspirin and other NSAID.
IV fluids should be given at a diuretic rate if the potential for renal damage exists. Alkalinization of the urine with sodium bicarbonate results in ion trapping of salicylates in kidney tubules and can increase their excretion. However, ion trapping should be used judiciously and only in cases where the acid-base balance can be monitored closely. Baseline renal function should be monitored and rechecked at 48 and 72 hr. Prognosis depends on the dose ingested and how soon the animal receives treatment following exposure."
http://www.merckvetmanual.com/mvm/index....
I agree with Ann.
What exactly did the vet do?
They should have induced vomiting and gave activated charcoal through a tube down her throat if it was caught in time.